ROM Resist & New Zealand

In this place and that place' the high gain thresholds are crossed by the local high velocities. [THAT is one long sentence stream!]. Short version: Speed kills. If true, then, changing the attachment such that low velocity becomes high velocity will make that BIG muscle BECOME fast & more spastic. Likewise, a uselessly nasty fast spastic muscle can be reclaimed to better function by shortening it's excursion. The two are not flip sides of the same coin as the natural high velocity muscles are typically small. Giving them a short lever attachment reduces both the spasticity and useful power.

The semitendinosus & gracilis have very long paths to insertion in the pes, well below knee center. These are ‘high speed ‘ muscles (distance/time) but small in actual muscle mass. They are, perhaps, more functional as sensory than as power sources. Thus they are both huge

sources of abnormal reflex when their spindles are allowed to run at top sensitivity undampened (as in spastic CP). The larger powerfulmuscles of small excursion, react less primarily but are recruited by the signal blast from the undampened spindles in the high speed muscles.