ROM Resist & New Zealand

The semitendinosus (ST) which well above the knee thins to a narrow tendon, had, at the surgery area, blended into a thick rope most of which was added to by the now conjoined semimembranosus (SM). ST is a smaller muscle with comparatively more tendon than muscle. It's tendon attaches far below the knee joint in a forward area called the 'pes'. Sports surgeons don't hesitate to steal the tendon (of the ST) to repair knee ligaments. Although some high speed control issues my be experienced in those that opt (bad <==Because of where it attaches (pes) any knee movement, at all, has a very long ST excursion. A lot of distance for any angular change. That = 'high velocity'. The SM is a huge muscle – very choice) to continue high performance competition.

powerful – which does the bulk of the work. It attaches right at the joint edge, nearly into the joint. Because it attaches so close to the knee joint, it has a really short length change with even full knee flexion. That = 'low velocity muscle'. ST and SM have the same muscle biology, but fast/slow relates to the attachment effect on distance traveled in a given arc and thus velocity. Rather than saying that muscles living 'in this place and that place' are more spastic, we can say that about muscles which have velocities that top out the spindle sensors (which is where spasticity actually lives) that are preset at too high gain (volume turned up too high).