SPMLs & EtOH

(though later we hear that super high performance athletes have some high speed fine control issues with this missing). OK. Just release the semitendinosus contracture. That was all we could feel holding him back. Like popping a balloon, his surgery was over in a matter of seconds with nice supple range. You already know it went quite well. But, the degree of response was way more generalized than what we did. Now go read the Range vs Resist book and have your mind blown. Mine was. It wasn’t range contracture that was disabling. It was what the resistance was eliciting (afar) that was disabling! It isn’t the range, the arc stop point, but the tension applied to what was NOT contracted that was disabling. A super high speed muscle is functionally removed (still there but not doing much) and spasticity goes away – including in other places. Smaller ranges are quite functional if what is left is stable and supple.

THAT is what is selective. Where is the pesky reaction coming from? Go there. Luck has it that the high velocity stuff is always near the surface where high speed lives. <== That means rapid movement is needed to find and measure how much SPML is enough, it done awake or fully reactive though sedated.

This selects anesthesia methods. What does not suppress reflex abnormality? For example, sevofluorane, a terrific anesthesia (fast in, fast out) totally suppresses abnormal reflexes and reflex spread. We can’t find what to fix nor how much under ‘sevo’.