Cerebral Palsy maybe/maybe not

That is shore front property. There is no blood flow crossing that ocean. Here all the vessels are kind of dead ends. Low pressure leaves this tissue wanting. Spotty bits undergo necrosis (croak) and at autopsy look like white (leuko) soft spots (malacia) in the peri-ventricular tissues, hence PVL. In horizontal cross section (exempli gratia in MRI) this zone looks like a boomerang shape with the apex pointing to the mid-line. This apex has the least flow and so determines the most frequent consequences. The foot- leg-hip tracts radiate away from this center. Trunk to hands are further from this arcing zone center. When you see hand involvement, the spread isn’t just longer along the boomerang region. The boomerang itself is thicker, especially spreading out at the apex. This then approaches neural hand areas and ‘ganglia’ which bring in some dystonic qualities – especially in the hands – less so in the legs. Given good oxygenation, but with low pressure caused low flow, the cortex is spared unless some of those extra vessels rupture (hemorrhage).

Hemorrhage? You know it as bleeding. If it bleeds into the ventricle then it is intraventricular hemorrhage.

Now it gets really interesting. Go take a vitamin. You’ll need it. We need to grapple with information theory & control. With this we need to consider that there are two functional ‘do it’ pathways, pyramidal and extra-pyramidal.

Pyramidal is the pathway you thought you already knew, the brain to muscle informational path [actually in cats, this isn’t quite so].

In most mammals and especially humans it is the telegraph line, hither to thither, with two transmission relays en route. So like pony express usually three horses to get the mail from here to there.